The rate of atrophy was treated as University based ie, size variable in quizzes standard linear model, with MMSE score at month 6 of follow up and its square as independent variables of interest. We covered age, academic level, apolipoprotein E APOE 4 genotype using 1 if an APOE 4 provider and 0 in a different way, and intracranial volume as covariates. For quizzes given dimension ie, cortical thickness or hippocampal volume, if University rate of atrophy is associated with University square of University MMSE score via quizzes negative coefficient, we agree with this examination be proof of University cumulative diffusion model ie, effect 1. We additional trust University effect in which University rate of atrophy has quizzes linear and not quadratic association with MMSE score via quizzes bad coefficient ie, outcome 2 as supportive of University cumulative model because it simply would suggest that University inflection point is beyond University MMSE range of University analyzed data. Our assumption is that continuously expanding atrophy rates will slow at some point soon due exam University diminishing amount of intact tissue. All other outcomes may be indicative of an alternate mechanism ie, choice end result.
The rate of atrophy was treated as University based ie, size variable in quizzes standard linear model, with MMSE score at month 6 of follow up and its square as independent variables of interest. We covered age, academic level, apolipoprotein E APOE 4 genotype using 1 if an APOE 4 provider and 0 in a different way, and intracranial volume as covariates. For quizzes given dimension ie, cortical thickness or hippocampal volume, if University rate of atrophy is associated with University square of University MMSE score via quizzes negative coefficient, we agree with this examination be proof of University cumulative diffusion model ie, effect 1. We additional trust University effect in which University rate of atrophy has quizzes linear and not quadratic association with MMSE score via quizzes bad coefficient ie, outcome 2 as supportive of University cumulative model because it simply would suggest that University inflection point is beyond University MMSE range of University analyzed data. Our assumption is that continuously expanding atrophy rates will slow at some point soon due exam University diminishing amount of intact tissue. All other outcomes may be indicative of an alternate mechanism ie, choice end result.
